Home /Products/Anti Estrogen Steroids/CAS 89778 27 8 Anti Estrogen Steroid Hormones White Raw Powder Toremifene Citrate Fareston
CAS 89778 27 8 Anti Estrogen Steroid Hormones White Raw Powder Toremifene Citrate Fareston
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CAS 89778 27 8 Anti Estrogen Steroid Hormones White Raw Powder Toremifene Citrate Fareston
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Basic Infomation
Place of Origin: China
Brand Name: YW
Certification: GMP, ISO9001, USP, KOSHER
Model Number: 89778-27-8
High Light:

natural estrogen blocker

,

natural anti estrogen supplements
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Packaging Details: Discreet package or as required
Delivery Time: 3~5 working days
Payment Terms: Western Union, Moneygram, Bitcoin and Bank wire transfer
Supply Ability: 5000kg/week
Specifications
Usage: Muscle Building
CAS: 89778-27-8
Assay: 98% Min.
Delivery: Express Courier.
Character: White Powder
MF: C32H36ClNO8
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Product Description
CAS 89778 27 8 Anti Estrogen Steroid Hormones White Raw Powder Toremifene Citrate Fareston for Breast Cancer Treatment
 
Description:
 
Fareston will display both estrogen antagonist / agonist properties in the body. This puts Fareston in the same category as Nolvadex and Clomid, the two most popular drugs in Farestons category. FARESTON is an estrogen agonist/antagonist indicated for the treatment of metastatic breast cancer in postmenopausal women with estrogen-receptor positive or unknown tumors.
 
Toremifene administration for a period of 3 months in men with idiopathic oligozoospermia is associated with significant improvements of sperm count, motility, and morphology, mediated by increased gonadotropin secretion and possibly a direct beneficial effect of toremifene on the testes. The above findings are also indicative of a better testicular exocrine (improved sperm parameters) response to treatment in men whose partners achieved pregnancy compared with those who did not. Further randomized, placebo-controlled trials should be conducted to determine whether this particular selective estrogen receptor modulator can be useful as an initial approach in men with oligozoospermia.
 
 
Quick details:
 
Toremifene Citrate
Alias: fareston;Toremifine
CAS: 89778-27-8
MF: C32H36ClNO8
Purity: 98.00%
Melting Point: 160-162°C
Boiling Point: 146-148C
density : 1,045g/cm
Storage temp: -20°C Freezer
Chemical Properties: White-to-Off-White Solid
Usage: antineoplastic.
 
 
Application:
 
Toremifene citrate is an oral selective estrogen receptor modulator (SERM) derived from triphenylethylene. It is FDA approved for breast cancer treatment, and has possible medical uses for prostate cancer. Bodybuilders will use this drug to combat gynecomastia (bitch tits), but it's still very new to the scene and has some drawbacks.
 
It has already been covered that Toremifen is a SERM, and serves to block Estrogen at various receptor sites in certain tissues within the body. As a layman explanation, Toremifen pretends to be a 'fake' Estrogen that occupies Estrogen receptors within breast tissue. With these receptors occupied by Toremifen, real Estrogen cannot perform their jobs there.
 
Toremifen does not reduce total blood plasma levels of Estrogen. In addition to being antagonistic to Estrogen receptors in breast tissue, it is also antagonistic to Estrogen at the hypothalamus gland this essentially 'tricks' the hypothalamus into thinking there is little or no circulating Estrogen levels in the body, causing it to increase its manufacture of Testosterone so that it can utilize aromatization to restore these levels.
 
Toremifen is also agonistic to Estrogen receptors in other tissues in the body. This means that whileToremifen will act as an anti-estrogen in breast tissue and the hypothalamus, it will act as an Estrogen within the liver. This can have beneficial impacts especially during an anabolic steroid cycle, such as improving and shifting cholesterol levels into a more favorable range.
 
 
COA:
 
TEST ITEMS SPECIFICATION RESULTS
Description White or Almost White Crystalline Powder White Powder
Identification Positive Positive
Assay 97.00~103.00% 99.38%
Loss On Drying 0.5%max 0.25%
Melting Point 95~99ºC 96.0~98.0ºC
Specific Rotation +48°~+51° +49.0°
Organic Volatile Impurities meets the requirements. Conforms
Residual Solvents meets the requirements. Conforms
Conclusion The specification conform with USP32 standard
 
 
Dosage:
 
The half life of fareston is around 5 days, which is approximately the same as clomid and nolvadex.For bodybuilding purposes,40-60mgs per day dosage seems to be a good starting point. If the user is trying to fight existing gynecomastia, they may choose to double the dose and add an aromatase inhibitor with it. For PCT, some users will kick-start with 100-120mgs per day and then lower to 40-60mgs per day for 4-6 weeks.
 
 
Toremifene Citrate Effect:
 
Toremifene citrate is an oral selective estrogen receptor modulator (SERM) which helps oppose the actions of estrogen in the body. Licensed in the United States under the brand name Fareston, toremifene citrate is FDA-approved for use in advanced (metastatic) breast cancer. It is also being evaluated for prevention of prostate cancer under the brand name Acapodene.
 
It has already been covered that Toremifene is a SERM, and serves to block Estrogen at various receptor sites in certain tissues within the body (breast tissue in particular). As a layman explanation, Toremifene pretends to be a ‘fake’ Estrogen that occupies Estrogen receptors within breast tissue. With these receptors occupied by Toremifene, real Estrogen cannot perform their jobs there. Toremifene does not reduce total blood plasma levels of Estrogen. In addition to being antagonistic to Estrogen receptors in breast tissue, it is also antagonistic to Estrogen at the hypothalamus gland (this essentially ‘tricks’ the hypothalamus into thinking there is little or no circulating Estrogen levels in the body, causing it to increase its manufacture of Testosterone so that it can utilize aromatization to restore these levels.
 
Toremifene Citrate exerts its effects by antagonizing the estrogen receptor in some tissues, and agonizing it in others. In this way, certain estrogenic pathways are activated and others are blockaded. It seems to exert estrogenic effects on blood lipids, reducing LDL and total cholesterol, as well as estrogenic effects on bone, improving density. It would also appear to exert anti-estrogenic effects in breast tissue, displacing the traditional effects of estrogen, effectively helping prevent breast cancer in postmenopausal women.

 

 

 

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